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Authors interpret these findings to mean that LR3 IGF-1-treated fetuses can effectively utilize the available nutrients and oxygen to support organ-specific growth, rather than IGF-1 infusion stimulating placental blood flow or nutrient transfer to the fetus
doi:10.1056/NEJMoa1908655
Heat shock protein 90 triggers multi-drug resistance of ovarian cancer via AKT/GSK3/-catenin signaling
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